Approximately 69 % of magnesium ions are stored in bone. the rest are part of the intermediary metabolism, about 70 % being present in free form while the other 30 % is bound to proteins (especially albumin), citrates, phosphate, and other complex formers. the mg 2+ serum level is kept constant within very narrow limits (0.65‑1.05 mmol/l. About 10 mmol of co 2 are produced per minute, (approximately 60 meq/l) when compared with other cells. most of the carbon dioxide (approximately 81%) is subsequently transported to the lung as bicarbonate. glucose is converted to two molecules of glyceraldehyde 3-phosphate that is subsequently converted to 1,3-bpg by glyceraldehyde 3. When hypocalcemia persists, it is best to delay calcium supplementation until the serum phosphate level is below 6 mg/dl to reduce the risk of metastatic calcification. 6) 0.5 mmol of elemental calcium = 1.0 meq. example conversion: 0.075 mmol elemental calcium/kg/hr = 0.15 meq/kg/hr = 3 mg/kg/hr. vitamin d: reference intakes / rda.
O potassium phosphate: 15 mmol/250 ml and 21 mmol/250 ml o sodium phosphate: 15 mmol/250 ml, 21 mmol/250 ml, and 30 mmol/250 ml current serum phosphorus level total phosphorus replacement monitoring 2 – 2.5 mg/dl 15 mmol potassium phosphate iv over 4 hr no additional action 1 – 1.9 mg/dl 21 mmol potassium phosphate iv over 4 hr recheck. Parenteral potassium phosphate contains 93 mg (3 mmol) phosphorus and 170 mg (4.4 meq or 4.4 mmol) potassium per ml. the usual dose is 0.5 mmol phosphorus/kg (0.17 ml/kg) iv over 6 hours. patients with alcohol use disorder may require ≥ 1 g/day during total parenteral nutrition; supplemental phosphate is stopped when oral intake is resumed.. Hypokalemia is serum potassium concentration < 3.5 meq/l (< 3.5 mmol/l) caused by a deficit in total body potassium stores or abnormal movement of potassium into cells. the most common cause is excess loss from the kidneys or gastrointestinal tract. clinical features include muscle weakness and polyuria; cardiac hyperexcitability may occur with severe hypokalemia..
Example: a 12-year-old boy with chronic renal failure (estimated gfr = 25.3 ml/min/1.73 m 2) aspirates after having a hypertension-induced seizure and is now intubated and on a ventilator.his blood gas shows a ph of 7.19, pco 2 of 46 mm hg, and hco 3 − of 18 mmol/l. his hemoglobin is 10.1 g/dl. the base excess is -10.8 meq/l. how much sodium bicarbonate per kilogram would be needed to. Sodium phosphate 15 to 30 mmol iv over 4-6hrs. it’s diluted in 250 ml of normal saline. ie. 0.08 to 0.24 mmol/kg over 6 hours (up to a maximum total dose of 30 mmol). recheck serum phosphorus level 2 hours after infusion complete < 1.25 mg/dl: sodium phosphate 30 mmol iv over 4-6hrs.. Once intracellular and extracellular concentrations are stable, a decrease in serum potassium concentration of about 1 meq/l (1 mmol/l) indicates a total potassium deficit of about 200 to 400 meq (200 to 400 mmol). patients with stable potassium concentration 3 meq/l (3 mmol/l) typically have a significant potassium deficit..
Sodium phosphate 15 to 30 mmol iv over 4-6hrs. it’s diluted in 250 ml of normal saline. ie. 0.08 to 0.24 mmol/kg over 6 hours (up to a maximum total dose of 30 mmol). recheck serum phosphorus level 2 hours after infusion complete < 1.25 mg/dl: sodium phosphate 30 mmol iv over 4-6hrs.. Parenteral potassium phosphate contains 93 mg (3 mmol) phosphorus and 170 mg (4.4 meq or 4.4 mmol) potassium per ml. the usual dose is 0.5 mmol phosphorus/kg (0.17 ml/kg) iv over 6 hours. patients with alcohol use disorder may require ≥ 1 g/day during total parenteral nutrition; supplemental phosphate is stopped when oral intake is resumed.. When hypocalcemia persists, it is best to delay calcium supplementation until the serum phosphate level is below 6 mg/dl to reduce the risk of metastatic calcification. 6) 0.5 mmol of elemental calcium = 1.0 meq. example conversion: 0.075 mmol elemental calcium/kg/hr = 0.15 meq/kg/hr = 3 mg/kg/hr. vitamin d: reference intakes / rda.
